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Rickets usually occurs because of a lack of vitamin D or calcium, although it can also be caused by a genetic defect or another health condition.
The most common cause of rickets is a lack of vitamin D or calcium in a child's diet. Both are essential for children to develop strong and healthy bones.
The main sources of vitamin D are:
Calcium is commonly found in dairy products, such as milk, cheese and yoghurt, and green vegetables, such as broccoli and cabbage.
Over time, a vitamin D or calcium deficiency will cause rickets in children and soft bones (osteomalacia) in adults.
See preventing rickets for more information and advice about ensuring your child gets enough vitamin D and calcium.
Any child who doesn't get enough vitamin D or calcium can develop rickets, but there are certain groups of children who are more at risk.
For example, rickets is more common in children of Asian, African-Caribbean and Middle Eastern origin because their skin is darker and needs more sunlight to get enough vitamin D.
Babies born prematurely are also at risk of developing rickets because they build up stores of vitamin D while they're in the womb. Babies who are exclusively breastfed, especially for longer than six months, may also be at risk of vitamin D deficiency.
As the amount of vitamin D in breast milk varies, the Department of Health recommends that all pregnant and breastfeeding women should take a daily supplement of 10 micrograms (µg) of vitamin D. This ensures the mother's vitamin D requirements are met and adequate foetal stores are built up for early infancy.
Rare forms of rickets can also occur in some inherited (genetic) disorders. For example, hypophosphatemic rickets is a genetic disorder where the kidneys and bones deal abnormally with phosphate.
Phosphate binds to calcium and is what makes bones and teeth hard. This leaves too little phosphate in the blood and bones, leading to weak and soft bones.
Other types of genetic rickets affect special proteins in the body that are used by vitamin D.
Occasionally, rickets develops in children with rare forms of kidney, liver and intestinal conditions. These can affect the absorption of vitamins and minerals.
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